EUROMEDICA  Hanover 1-2  Juni 2007	Advanced methods of diagnosis, treatment and prophylactics
	European Academy of Natural Sciences, Hanover European Scientific Society, Hanover Russian Academy of Natural Sciences, Moscow

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The Myofascial Pain Syndrome (MPS) is a frequent complication among patients who had Cerebral Stroke. We found a clinical variation of this syndrome, its frequency of occurrence, typical trigger localization and period of onset.

The MPS affects 85, 9 % of patients after a Cerebral Stroke, but can reveal itself at different times. 54, 1 % of patients show signs of MPS at the acute period; 14, 8 % during early rehabilitation; 13, 9 % of patients during later rehabilitation and 17, 2 % of patients with the consequences of the disease among the checked up patients.

The Pain Syndrome appears in the first weeks of the Stroke along with spasticity increasing in paralyzed extremities (in 8,3 ± 1,9 days). Triggers localized in upper extremities affect the shoulder girdle muscle, biceps and deltoid regions. Lower paretic extremities involve the fore-surface of thighs. But only 37, 7 % of patients have the Pain Syndrome in lower extremities. There are often two, three or more active triggers that appeared in paretic extremities. Intensity of trigger activity symptoms varied from a low of 16 % to a high of 84 %.

In 82, 6 % of the medical observation the high intensity of muscular traction at the affected by the trigger muscle was found. Pronounced extend of local convulsive response was 88, 1 %.

In 54, 5 % of triggers in the upper extremities appeared a zone of deep painfulness with vegetative displays, so called “Reflected zone”. The reflected zone is mostly localized in the anteriomedialis surface of the forearm and in the radiocarpal articulation. For the lower extremities, the zone of reflected painfulness is localized in the interiomedalis surface of the shins and feet.

The authentic correlated connection between trigger quantity and pain intensity of triggers with the period of disease (r = 0, 344) and spasticity (r = 0, 670) was found. The trigger quantity correlated with the extent of paresis (r = 0, 072).

The correlation of trigger pain extent and their quantity, with the existence and extent of sensible abnormalities, was not found.